From b729a07499b32b4b37d7f6574f897cc5fea6ee40 Mon Sep 17 00:00:00 2001 From: LianeHughes Date: Fri, 17 May 2024 12:33:52 +0200 Subject: [PATCH] Update content/english/highlights/receptor_incompatability_infa.md Co-authored-by: Senthilkumar Panneerselvam --- content/english/highlights/receptor_incompatability_infa.md | 2 +- 1 file changed, 1 insertion(+), 1 deletion(-) diff --git a/content/english/highlights/receptor_incompatability_infa.md b/content/english/highlights/receptor_incompatability_infa.md index e939ab8e..2eb631ff 100644 --- a/content/english/highlights/receptor_incompatability_infa.md +++ b/content/english/highlights/receptor_incompatability_infa.md @@ -19,7 +19,7 @@ tags: zoonosis, ] authors: [Katarina Öjefors Stark] -images: [/highlights/banners/infA_receptors_large.jpg"] +images: [/highlights/banners/infA_receptors.jpg"] --- When a novel variant of the influenza A virus (IAV) emerges, it results in an influenza outbreak if immunity within human population is low. Rapid rates of interpersonal transmission and high rates of infection can result in such an IAV becoming a global pandemic. Studies have indicated that the three most recent IAV pandemics (H2N2 1957 Asian flu, H3N2 1968 HongKong flu, and H1N1 2009 Swine flu) were all caused by zoonotic viruses; they all contain genetic material from avian influenza viruses (AIVs). The exact transmission route from bird to human remains unclear, but each of the pandemics probably involved spread from wild to domestic bird species, and then to mammals (e.g. pigs and humans).