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ADD: retrocopy.rst: Complete references
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References and Further Reading
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.. [1] McCLINTOCK, 1950.
.. [2] BURNS, 2017.
.. [3] DE KONING et al., 2011.
.. [4] KAESSMANN, 2010.
.. [5] HELMAN et al., 2014.
.. [6] LANDER et al., 2001.
.. [7] HANCKS and KAZAZIAN, 2016.
.. [8] DENLI et al. 2015.
.. [9] BATZER and DEININGER, 2002.
.. [10] KAESSMANN et al., 2009.
.. [11] DEININGER, 2011.
.. [12] BAKSHI et al. 2016.
.. [13] BECK et al., 2010.
.. [14] LEE et al., 2012.
.. [15] NAVARRO and GALANTE, 2013.
.. [16] NAVARRO and GALANTE, 2015.
.. [17] MIKI et al., 1992.
.. [18] SOLYOM et al., 2012.
.. [19] COOKE et al., 2014.
.. [20] SUNAMI et al. 2011.
.. [21] HUR et al., 2014.
.. [22] CLAYTON et al. 2016
.. [23] DEININGER and BATZER, 1999.
.. [24] BELANCIO et al. 2010.
.. [25] POLISENO et al, 2010.
.. [1] MCCLINTOCK, B. (1950).
The origin and behavior of mutable loci in maize.
Proceedings of the National Academy of Sciences of the United States of America,
36(6), 344–355.
.. [2] BURNS, K. H. (2017).
Transposable elements in cancer.
Nature reviews. Cancer, 17(7), 415–424.
.. [3] DE KONING, A. P. J. et al. (2011).
Repetitive Elements May Comprise Over Two-Thirds of the Human Genome.
PLoS genetics, 7(12), e1002384.
.. [4] KAESSMANN, H. (2010).
Origins, evolution, and phenotypic impact of new genes.
Genome research, 20(10), 1313–1326.
.. [5] HELMAN, E. et al. (2014).
Somatic retrotransposition in human cancer revealed by whole-genome and exome sequencing.
Genome research, 24(7), 1053–1063.
.. [6] LANDER, E. S. et al. (2001).
Initial sequencing and analysis of the human genome.
Nature, 409(6822), 860–921.
.. [7] HANCKS, D. C. and KAZAZIAN, H. H. (2016).
Roles for retrotransposon insertions in human disease.
Mobile DNA, 7(9).
.. [8] DENLI, A. M. et al. (2015).
Primate-specific ORF0 contributes to retrotransposon-mediated diversity.
Cell, 163(3), 583–593.
.. [9] BATZER and DEININGE. (2002).
Alu repeats and human genomic diversity.
Nature reviews. Genetics, 3(5), 370–379.
.. [10] KAESSMANN, H. et al. (2009).
RNA-based gene duplication: mechanistic and evolutionary insights.
Nature reviews. Genetics, 10(1), 19–31.
.. [11] DEININGER, P. (2011).
Alu elements: know the SINEs.
Genome biology, 12(12), 236.
.. [12] BAKSHI et al. (2016).
DNA methylation variation of human-specific Alu repeats.
Epigenetics: official journal of the DNA Methylation Society, 11(2), 163–173.
.. [13] BECK et al. (2010).
LINE-1 retrotransposition activity in human genomes.
Cell, 141(7), 1159–1170.
.. [14] LEE, E. et al. (2012).
Landscape of somatic retrotransposition in human cancers.
Science, 337(6097), 967–971.
.. [15] NAVARRO, F. C. P. and GALANTE, P. A. F. (2013).
RCPedia: a database of retrocopied genes.
Bioinformatics, 29(9), 1235–1237.
.. [16] NAVARRO, F. C. P. and GALANTE, P. A. F. (2015).
A Genome-Wide Landscape of Retrocopies in Primate Genomes.
Genome biology and evolution, 7(8), 2265–2275.
.. [17] MIKI, Y. et al. (1992).
Disruption of the APC gene by a retrotransposal insertion of L1 sequence in a colon cancer.
Cancer research, 52(3), 643–645.
.. [18] SOLYOM, S. et al. (2012).
Extensive somatic L1 retrotransposition in colorectal tumors.
Genome research, 22(12), 2328–2338.
.. [19] COOKE, S. L. et al. (2014).
Processed pseudogenes acquired somatically during cancer development.
Nature communications, 5, 3644.
.. [20] SUNAMI, E. et al. (2011).
LINE-1 hypomethylation during primary colon cancer progression.
PloS one, 6(4), e18884.
.. [21] HUR, K. et al. (2014).
Hypomethylation of long interspersed nuclear element-1 (LINE-1) leads to
activation of proto-oncogenes in human colorectal cancer metastasis.
Gut, 63(4), 635–646.
.. [22] CLAYTON, E. A. et al. (2016).
Patterns of Transposable Element Expression and Insertion in Cancer.
Frontiers in molecular biosciences, 3, 76.
.. [23] DEININGER, P. L. and BATZER, M. A. (1999).
Alu repeats and human disease.
Molecular genetics and metabolism, 67(3), 183–193.
.. [24] BELANCIO et al. (2010).
All y’all need to know 'bout retroelements in cancer.
Seminars in cancer biology, 20(4), 200–210.
.. [25] POLISENO, L. et al, (2010).
A coding-independent function of gene and pseudogene mRNAs regulates tumour biology.
Nature, 465(7301), 1033–1038.

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