Update tsv, md and png files

_pathways/WP3995.md

@@ -12,14 +12,6 @@ annotations:
   parent: disease pathway
   type: Pathway Ontology
   value: disease pathway
-- id: CL:0000540
-  parent: animal cell
-  type: Cell Type Ontology
-  value: neuron
-- id: CL:0000540
-  parent: animal cell
-  type: Cell Type Ontology
-  value: neuron
 authors:
 - LvdWouw
 - Fehrhart
@@ -57,15 +49,15 @@ description: 'Prion diseases are rare, genetic, transmissible and sporadic disea
   transcription of genes which are important for axonal growth, survival, and synaptic
   plasticity in neurons.  Proteins on this pathway have targeted assays available
   via the [https://assays.cancer.gov/available_assays?wp_id=WP3995 CPTAC Assay Portal]'
-last-edited: 2024-02-05
+last-edited: 2024-04-25
 ndex: ff8e2fc6-8b68-11eb-9e72-0ac135e8bacf
 organisms:
 - Homo sapiens
 redirect_from:
 - /index.php/Pathway:WP3995
 - /instance/WP3995
-- /instance/WP3995_r128423
-revision: r128423
+- /instance/WP3995_r129475
+revision: r129475
 schema-jsonld:
 - '@context': https://schema.org/
   '@id': https://wikipathways.github.io/pathways/WP3995.html

_pathways/WP5258.md

@@ -1,5 +1,13 @@
 ---
 annotations:
+- id: PW:0001028
+  parent: disease pathway
+  type: Pathway Ontology
+  value: infectious disease pathway
+- id: PW:0000013
+  parent: disease pathway
+  type: Pathway Ontology
+  value: disease pathway
 - id: DOID:0080600
   parent: disease by infectious agent
   type: Disease Ontology
@@ -13,6 +21,7 @@ authors:
 - Egonw
 - Eweitz
 - Khanspers
+- Mkutmon
 citedin: ''
 communities: []
 description: Proposed mechanism for COVID-19 induced endothelial dysfunction via ROS,
@@ -21,15 +30,15 @@ description: Proposed mechanism for COVID-19 induced endothelial dysfunction via
   of functional ACE2, which leads to increased ROS via imbalanced RAS signaling. Excessive
   ROS disturbs vascular tone and increases endothelial permeability.  Description
   modified from [https://europepmc.org/article/MED/34838588 Ma et. al.].
-last-edited: 2024-02-10
+last-edited: 2024-04-25
 ndex: null
 organisms:
 - Homo sapiens
 redirect_from:
 - /index.php/Pathway:WP5258
 - /instance/WP5258
-- /instance/WP5258_r128499
-revision: r128499
+- /instance/WP5258_r129476
+revision: r129476
 schema-jsonld:
 - '@context': https://schema.org/
   '@id': https://wikipathways.github.io/pathways/WP5258.html
@@ -58,8 +67,8 @@ schema-jsonld:
   - TNFRSF1A
   - nitric oxide
   license: CC0
-  name: Role of ROS in COVID-19 endothelial dysfunction
+  name: ROS in COVID-19 endothelial dysfunction
 seo: CreativeWork
-title: Role of ROS in COVID-19 endothelial dysfunction
+title: ROS in COVID-19 endothelial dysfunction
 wpid: WP5258
 ---

_pathways/WP5304.md

@@ -1,5 +1,9 @@
 ---
 annotations:
+- id: CL:0000182
+  parent: native cell
+  type: Cell Type Ontology
+  value: hepatocyte
 - id: PW:0001346
   parent: regulatory pathway
   type: Pathway Ontology
@@ -16,19 +20,23 @@ authors:
 - TomPauly
 - Susan
 - Egonw
+- Eweitz
+citedin: ''
+communities: []
 description: 'This pathway includes the following processes regarding cholesterol
   metabolism: I) Absorption of cholesterol in the enterocyte, II) Forming of chylomicrons
   and other lipid droplets, III) Distinction between Bloch and Kandutsch-Russell cholesterol
   biosynthesis, IV) Absorption of cholesterol in the hepatocyte and V) Transintestinal
   cholesterol excretion (TICE).'
-last-edited: 2023-01-29
+last-edited: 2024-04-25
+ndex: null
 organisms:
 - Homo sapiens
 redirect_from:
 - /index.php/Pathway:WP5304
 - /instance/WP5304
-- /instance/WP5304_r125192
-revision: r125192
+- /instance/WP5304_r129477
+revision: r129477
 schema-jsonld:
 - '@context': https://schema.org/
   '@id': https://wikipathways.github.io/pathways/WP5304.html